Introduction
Lateral medullary syndrome (LMS) is a neurological disease caused by the
lesion of the lateral part of the medulla oblongata.1Dysphagia, a common complication of LMS, is clinically important because
of its associations with aspiration pneumonia, malnutrition, increased
mortality, and decreased quality of life. Previous studies have
suggested that the swallowing center, which includes the nucleus
ambiguus (NA) and the nucleus tractus solitarius (NTS), coordinates the
pharyngeal phases of swallowing. Fujishima et al. reported UES and
pharyngeal incoordination during swallowing in dysphagia patients with
LMS using conventional sensor.2 However, the
pathophysiology of dysphagia has not been fully elucidated in these
patietns.1,2
Herein, we present a case in which
high-resolution manometry (HRM) was used to examine the pathophysiology
of a patient with severe bulbar dysphagia due to LMS.