Introduction
Lateral medullary syndrome (LMS) is a neurological disease caused by the lesion of the lateral part of the medulla oblongata.1Dysphagia, a common complication of LMS, is clinically important because of its associations with aspiration pneumonia, malnutrition, increased mortality, and decreased quality of life. Previous studies have suggested that the swallowing center, which includes the nucleus ambiguus (NA) and the nucleus tractus solitarius (NTS), coordinates the pharyngeal phases of swallowing. Fujishima et al. reported UES and pharyngeal incoordination during swallowing in dysphagia patients with LMS using conventional sensor.2 However, the pathophysiology of dysphagia has not been fully elucidated in these patietns.1,2
Herein, we present a case in which high-resolution manometry (HRM) was used to examine the pathophysiology of a patient with severe bulbar dysphagia due to LMS.