ACKNOWLEDGMENTS
This work was supported by grants from the National Natural Science
Foundation of China (NSFC, 81572542 and NSFC, 81874096), and Research
Project of Key Discipline of Guangdong Province (2019GDXK0010), Science
and Technology Program of Guangzhou City (The roles of AR mitochondrial
translocation in the development of anti-androgen drug resistance in
prostate cancer), National Key Specialty Construction Project of
Clinical Pharmacy, High Level Clinical Key Specialty of Clinical
Pharmacy in Guangdong Province.
ACKNOWLEDGEMENTS