ACKNOWLEDGMENTS
This work was supported by grants from the National Natural Science Foundation of China (NSFC, 81572542 and NSFC, 81874096), and Research Project of Key Discipline of Guangdong Province (2019GDXK0010), Science and Technology Program of Guangzhou City (The roles of AR mitochondrial translocation in the development of anti-androgen drug resistance in prostate cancer), National Key Specialty Construction Project of Clinical Pharmacy, High Level Clinical Key Specialty of Clinical Pharmacy in Guangdong Province.
ACKNOWLEDGEMENTS