The mechanism of PH₃ toxicity is through an irreversible metabolic crisis and/or the indirect effect of increased oxidative stress (16) (17) (18). The formation of reactive oxygen species (ROS), causes lipid peroxidation and protein denaturation, with severe cellular damage (19) (20) (21). In patients with severe acute AlP poisoning, cardiotoxicity is the main cause of morbidity and mortality (22) (23) (24). The reported mortality rate in ingestions ranges from 31 % (13) to 91% (25).